Tiff-Dysplasia: THE START OF MY JOURNEY!!!

Tuesday, August 25, 2009

THE START OF MY JOURNEY!!!

This is the start of my journey!!! I will be posting more in the coming days about my diagnosis and the emotions that follow thank you for checking out my blog!!!!!

1 comment:

HipSk8August 25, 2009 at 6:58 PM
Tiffany - To answer your question ... I have a post which lists what I found useful at the hospital:

http://hipsk8.blogspot.com/2009/07/lists.html

Cassie also has a list of things she took to the hospital on her blog:

http://hip-hopanonymous.blogspot.com/2008/10/in-my-hospital-bag.html

Everyone is different but I hope you find these useful. Terri
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HELPFUL DEFINITIONS

Hip dysplasia
Hip dysplasia is a congenital condition in which the ball and socket joint don't fit together the way they should. In my case, the socket is too shallow, which causes uneven pressure on the joint, a premature breakdown of cartilage and early osteoarthritis.
PAO surgery
involves cutting the pelvis in a few places and screwing it back together in a way that makes the socket joint less shallow and more accepting of the ball joint. It's meant to preserve the hip and prevent further arthritis and pain. A PAO is not a hip replacement, which is not ideal for people my age because they only last 10 years or so and require additional surgeries for revisions.
Femoroacetabular impingement (FAI)
previously termed acetabular rim syndrome, is a major factor in early development of osteoarthritis of the hip. This syndrome is caused by abnormal contact between the osseous protrusions of the acetabulum and the femur during hip joint movement.1 In patients with femoroacetabular impingement, repetitive microtrauma from the osseous impingement leads to recurring irritation, degenerative changes of the labrum, and ultimately irreversible cartilaginous damage. Femoroacetabular impingement tends to involve young, active patients and can be debilitating. Femoroacetabular impingement is often bilateral, but may be symptomatic on only one side. Femoroacetabular impingement is diagnosed and classified into two types, pincer and cam, by the clinical and radiologic presentation. However, most cases of femoroacetabular impingement are a combination of the two subtypes.Pincer impingement is caused by acetabular overcoverage with normal femoral head sphericity. The pincer effect is the linear contact between a normal neck and an overcovering acetabular rim, which can lead to labral damage. Pincer impingement is more common in middle-aged women with abnormalities of acetabular morphology.
Hip Labral Tear
The other name of hip labral tear is Acetabular Labral Tear. Labral tears frequently takes place in the front and top segments of the labrum. Hip labral (labrum) tear is a medical condition within the hip where a sort of cartilage takes place. The damage to the cartilage inside the socket of the hip joint makes your thighbone less stable inside the joint. Because of this complication the fluid may leak from the joint which may result with a friction in your hip. The cartilage is important for the hip joint for stability. The cartilage cushions the hip joint providing mobility to a person by giving autonomy and flexibility for movement.
Heterotopic Ossification
is the process by which bone tissue forms outside of the skeleton. Heterotopic ossifications will develop in 10% to 80% of cases with varying severity after surgery or trauma to the hip and lower legs. About every third patient who has to face heterotopic ossification following total hip arthoplasty (replacement) or severe fracture of the long bones of the lower extremities will develop pain and dysfunction resulting from extensive heterotopic ossification. Heterotopic ossification jeopardizes functional outcome, impairs rehabilitation and is costly because of secondary surgical procedures. Pain first arises a few days after surgery with calcified structures appearing as blurred contours on x-rays at 3 to 6 weeks postoperatively. Patients with heterotopic ossification after a previous hip arthoplasty are at greatest risk of developing additional heterotopic ossification, with incidence between 50% and 90%